Dr bruetsch in this collection of reprints has demonstrated a well known fact that a late sequel of rheumatic fever is an obliterating endarteritis which may involve the small meningeal and cortical vessels resulting in infarction of parts of the central nervous system. Clinical manifestations fever it is usually present during first week to ten days of rheumatic fever and is rarely above 39 0 cif patients temperature elevation persistently exceeds 39 0 c other cause should be sought in particular in the presence of rheumatic heart disease superimposed infective endocarditis should be the first consideration. Molecular mimicry between the group a streptococcus heart and brain is supported by evidence from studies of human mabs human t cell clones and serum igg antibodies derived from streptococcal sequelae and rheumatic fever galvin hemric ward cunningham 2000 kirvan swedo heuser cunningham 2003 human mabs derived from rheumatic carditis and sydenham chorea have supported the . Rheumatic fever is an inflammatory disease that may develop two to three weeks after a group a streptococcal infection such as strep throat or scarlet fever it is believed to be caused by antibody cross reactivity and can involve the heart joints skin and brain when the brain is damaged due
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